Oxidative-stress sensor (OxyR + CRISPRa)

An ingestible sensor for hydrogen peroxide / oxidative stress, a marker of gut inflammation. The native OxyR peroxide sensor gates a CRISPRa circuit driving a fluorescent reporter, in the probiotic E. coli Nissle 1917.

Clinical / gut biomarkerBSL-1 chassistemplateclinical-gutinflammationhydrogen-peroxideoxidative-stressprobioticNissleCRISPRa
Input
Hydrogen peroxide (H2O2) / oxidative stress
Clinical / gut biomarker
Sense
CRISPRa-activation
dCas9-ω (CRISPRa activator)
Chassis
E. coli Nissle 1917
BSL-1
Output
sfGFP
fluorescent

What it detects

Analyte
Hydrogen peroxide (H2O2) / oxidative stress — OxyR responds to low-µM H2O2
Category
Clinical / gut biomarker
Signal
Luminal hydrogen peroxide and reactive oxygen produced during gut inflammation

Genetic circuit

⤢ click to enlarge

Genetic construct (SBOL)

The DNA construct as transcription units, drawn with SBOL Visual part glyphs.

⤢ click to enlarge

CRISPR sensing mechanism

Strategy
CRISPRa-activation · amplifier logic
Cas protein
dCas9-ω (CRISPRa activator)
Analyte sensor
OxyR is oxidized by H2O2 and, in its oxidized form, activates antioxidant promoters such as PkatG / PahpC.
Signal flow
H2O2 -> oxidized OxyR activates PahpC -> transcribes an sgRNA -> dCas9-activator amplifies a fluorescent reporter (CRISPRa) -> fluorescence reports oxidative stress.

Safe chassis

E. coli Nissle 1917Escherichia coli

A probiotic E. coli used in humans for over a century (Mutaflor). Colonizes the gut safely, making it the chassis of choice for clinical / gut biomarker biosensors.

BSL-1probiotic

Genetic parts

PartRoleSource / id
OxyR regulator
H2O2-responsive transcriptional activator; endogenous to E. coli Nissle.
regulatorNative E. coli oxyR
PahpC / PkatG promoter
Activated by oxidized OxyR.
promoterE. coli OxyR-regulated antioxidant promoter
Reporter-activating sgRNAsgRNAdesigned for CRISPRa upstream of a weak reporter promoter
sgRNA scaffold (SpCas9)
GTTTTAGAGCTAGAAATAGCAAGTTAAAATAAGGCTAGTCCGTTATCAACTTGAAAAAGTGGCACCGAGTCGGTGC
sgRNAStandard SpCas9 scaffold
dCas9-ω activatordCas9Bikard et al. 2013 (CRISPRa)
sfGFP reporter
Recoverable from stool for non-invasive readout.
reporterPedelacq et al. 2006

Output & readout

Type
fluorescent
Reporter
sfGFP
Readout
Fluorescence (flow cytometry on recovered cells)
Positive result
Fluorescent-cell fraction rises with luminal oxidative stress.

Performance

Limit of detection
OxyR module: low-µM H2O2 (module-validated).
Dynamic range
~1-100 µM H2O2
Response time
~120 min
Device validated
No — design template (parts validated individually)

OxyR peroxide sensing and CRISPRa are validated separately; integrated device is a design template. Complements the thiosulfate/tetrathionate inflammation sensors with an oxidative-stress marker.

Safety

Biosafety level
BSL-1 (non-pathogenic chassis)
GRAS chassis
No
Biocontainment
Probiotic E. coli Nissle host; add thyA/dapA auxotrophy for gut-restricted containment.
Field-deployable
Lab / supervised use

Probiotic chassis with a human-safety record; research / supervised clinical use only.

Build & run

#StageStep
1designDesign CRISPRa sgRNA
Target the activation window of a weak reporter promoter; check Nissle off-targets.
2assemblyAssemble units
TU1: PahpC -> sgRNA (native OxyR). TU2: dCas9-omega. TU3: weak promoter -> sfGFP. Use a stable low-copy vector.
3transformationTransform E. coli Nissle 1917
Select; add auxotrophic containment.
4inductionValidate with H2O2
Confirm peroxide-dependent activation across a standard curve before any animal work.
5readoutRecover and measure
Recover cells from stool; quantify fluorescence.

Source & parts

Design
Design template combining the native OxyR peroxide sensor with a dCas9 CRISPRa circuit in E. coli Nissle
Parts validated in
  • Storz / Imlay OxyR peroxide-sensing literature
  • Bikard et al. 2013, NAR (CRISPRa)
License
Parts per their original sources; design template CC BY 4.0